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Calcibloc (Nifedipine)

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Calcibloc is used to treat hypertension (high blood pressure) and angina (chest pain). Calcibloc is in a class of drugs called calcium channel blockers. Calcibloc relaxes (widens) your blood vessels (veins and arteries), which makes it easier for the heart to pump and reduces its workload.

Other names for this medication:
Amarkor, Aprical, Buconif, Calcibloc, Calcigard, Cardifen, Citilat, Cordafen, Corotrend, Depicor, Farmalat, Ficard, Hexadilat, Jutadilat, Macorel, Menoprizin, Myogard, Nelapine, Nifatenol, Nifcal, Nifecap, Nifecard, Nifeclair, Nifedalat, Nifedical, Nifedicor, Nifedigel, Nifedip, Nifedipin, Nifedipina, Nifehexal, Nifelat, Nifelong, Nifestad, Niften, Nilol, Nipidin, Nipin, Nipress, Noviken, Ospocard, Pabalat, Tensomax, Valni, Vasdalat, Viscard

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Also known as: Nifedipine


Nifedipine is used alone or together with other medicines to treat severe chest pain (angina) or high blood pressure (hypertension). High blood pressure adds to the workload of the heart and arteries. If it continues for a long time, the heart and arteries may not function properly. This can damage the blood vessels of the brain, heart, and kidneys, resulting in a stroke, heart failure, or kidney failure. High blood pressure may also increase the risk of heart attacks. These problems may be less likely to occur if blood pressure is controlled.

Nifedipine is a calcium channel blocker. It works by affecting the movement of calcium into the cells of the heart and blood vessels. As a result, nifedipine relaxes blood vessels and increases the supply of blood and oxygen to the heart while reducing its workload.

This medicine is available only with your doctor's prescription.


This section provides information on the proper use of a number of products that contain nifedipine. It may not be specific to Adalat XL. Please read with care.

In addition to the use of this medicine, treatment for your high blood pressure may include weight control and changes in the types of foods you eat, especially foods high in sodium (salt). Your doctor will tell you which of these are most important for you. You should check with your doctor before changing your diet.

Many patients who have high blood pressure will not notice any signs of the problem. In fact, many may feel normal. It is very important that you take your medicine exactly as directed and that you keep your appointments with your doctor even if you feel well.

Remember that this medicine will not cure your high blood pressure, but it does help control it. You must continue to take it as directed if you expect to lower your blood pressure and keep it down. You may have to take high blood pressure medicine for the rest of your life. If high blood pressure is not treated, it can cause serious problems such as heart failure, blood vessel disease, stroke, or kidney disease.

Swallow the extended release tablet whole. Do not break, crush, or chew it. It is best to take this tablet on an empty stomach.

If you are taking the extended-release tablets, part of the tablet may pass into your stool after your body has absorbed the medicine. This is normal and nothing to worry about.

The dose of this medicine will be different for different patients. Follow your doctor's orders or the directions on the label. The following information includes only the average doses of this medicine. If your dose is different, do not change it unless your doctor tells you to do so.

The amount of medicine that you take depends on the strength of the medicine. Also, the number of doses you take each day, the time allowed between doses, and the length of time you take the medicine depend on the medical problem for which you are using the medicine.

For treatment of chest pain or high blood pressure: For oral dosage form (capsules): Adults—At first, 10 milligrams (mg) three times a day. Your doctor may increase your dose as needed. Children—Use and dose must be determined by your doctor. For oral dosage form (extended-release tablets): Adults—At first, 30 or 60 milligrams (mg) once a day. Your doctor may increase your dose as needed. However, the dose is usually not more than 90 mg once a day. Children—Use and dose must be determined by your doctor.


If you overdose Calcibloc and you don't feel good you should visit your doctor or health care provider immediately.


Store the medicine in a closed container at room temperature, away from heat, moisture, and direct light. Keep from freezing.

Keep out of the reach of children.

Do not keep outdated medicine or medicine no longer needed.

Ask your healthcare professional how you should dispose of any medicine you do not use.

Detailed Nifedipine dosage information

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


Tell your doctor if you have ever had any unusual or allergic reaction to this medicine or any other medicines. Also tell your health care professional if you have any other types of allergies, such as to foods, dyes, preservatives, or animals. For non-prescription products, read the label or package ingredients carefully.

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On the pattern of changes in the rat nervous system produced by 2,5 hexanediol. The changes in energy charge of the nematodes which occurred following administration of levamisole to their hosts was of the order which can be satisfactorily explained by changes in the environment of the nematodes, such as reduced oxygen tension. The synergistic effect was mediated through inhibition of COX-2. Radioimmunolocalization of metastatic breast carcinoma using indium-111-methyl benzyl DTPA BrE-3 monoclonal antibody: phase I study. There is a very plausible biologic mechanism to explain the relationship. The thymus plays the major role in the development of self-tolerance, and initial work in the late 1960s indicated that the thymus also plays a critical role in the induction of acquired tolerance to exogenous antigens.

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We report on the use of 125I and 131I labeling and of new, multicolor, multi-photon detection (MPD) methods to routinely and quantitatively detect protein spots on two-dimensional gel electrophoresis plates in the zeptomole to attomole range. At 6 weeks after transplantation, cardiac grafts were evaluated by histopathologic analysis in terms of vasculopathy, fibrosis, and cellular infiltration. Disorders of fetoplacental function in pregnant women with diabetes. They can live in soil, water supplies, plants and various environmental conditions. The main finding is that during the allowed social interactions, the dominant ND-pretreated rats spent more time on highly aggressive behaviors than the dominant placebo-treated rats. Biochemical characterization of ethanol actions on dihydropyridine-sensitive calcium channels in brain synaptosomes. Chromosomal mapping of the human interleukin-1 receptor antagonist gene (IL-1RN) and isolation of specific YAC clones. In all cases, a clinical grading system was used to assess the severity of inflammation before imaging. The incidence of atrial fibrillation (AF) increases with aging, but the aging-associated electrophysiological changes of atrial myocardium are poorly understood.

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The effects of varying dosages of irradiation on benign and malignant pelvic lymph nodes. Thrombin activatable fibrinolysis inhibitor (TAFI) polymorphisms and plasma TAFI levels measured with an ELISA insensitive to isoforms in patients with venous thromboembolic disease (VTD). To evaluate if the immunofluorescence analysis of synovial tissue (ST) using antibodies against RANKL/OPG, conjugated with the immunophenotyping of lymphocytes and macrophages, could be of diagnostic and prognostic value in rheumatoid arthritis (RA) patients. Gamma-aminobutyric acid (GABA)(B)-receptor antagonist CGP35348 was injected intracerebroventricularly (icv). Melanin-concentrating hormone (MCH) is a cyclic peptide which is predominantly synthetized in the hypothalamus of fish and mammalian brains.

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Urodynamic studies are unable to answer this question, as the instantaneous urethral resistance is evaluated globally by the Pressure-Flow relation. Detecting molecular interactions that stabilize native bovine rhodopsin. On the anastomoses of the coronary arterial circulation: comparative observations and considerations. In the present study, we evaluated the expression of VEGF in hepatic warm ischemia-reperfusion (I/R) injury and examined the effect of recombinant risperdal dosage injection human (rh)VEGF administration in an established murine model. Data from the Follow-up Survey of the Ontario Child Health Study were used. The objectives of this study was to investigate the 1-year recurrence rate of primary spontaneous pneumothorax after treatment by simple aspiration and the potential risk factors for recurrence. Root responses and metal accumulation in two contrasting ecotypes of Sedum alfredii Hance under lead and zinc toxic stress.

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Effect of magnesium deficiency and excess on renal tubular potassium transport in the rat. In particular, we argue that the size of the initial network core and its density of connections may be the main responsible for the exponential truncation of the power-law behavior observed in some small scale-free networks. Because the presence of FGF-positive RPE cells coincides temporally with increased nuclear thymidine labeling in the RPE and choriocapillaris, aFGF and/or bFGF may be at least partly responsible for initiating the process of cell proliferation and subretinal neovascularization in these animals. Immunohistochemical analysis reveals high frequency of PMS2 defects in colorectal cancer. A wide spectrum of microbes has acquired elegant mechanisms to overcome plavix drug interactions or deflect the host responses mediated by TNF.

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Questions regarding the distribution of stress in the proximal human femur have never been adequately resolved. The inhibition rate constants were at least 10 times higher than the pairing rate constants. A total of 20 teams consisting of three registered nurses and one resident each. Two mitochondrial enzymes, succinate dehydrogenase (SDH), a prominent enzyme in the Krebs cycle, and alpha-glycerophosphate dehydrogenase (alpha-GPDH), half of the glycerophosphate shuttle system for bringing reducing equivalents from cytoplasm to mitochondria, were examined enzyme histochemically to assess the contribution of each to metabolism of the developing canine retina. Spermine oxidase activity could be detected in fetal calf serum (FCS), and six major spermine oxidation products were revealed in the incubation mixture. Nitric oxide may be involved in this penile erection-inducing effect.

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Cell fusion of embryonic chick myoblasts has been studied in the presence of fat-soluble agents that induce erythrocytes to fuse. This cystatin classification should facilitate the assignment of proteinase specificities and functions to other cystatins as new information is obtained. It is shown that the new vessels appear as a reaction of the tissues hypoxemia and especially to alon of the oxygen pressure that determines the increase of the vessels. In particular there is a predominance of charged and polar residues in the region of the TCRA transcript thought to interact with peptide. (Diptera: Muscidae), caused by dietary gamma-aminobutyric acid (GABA). The authors describe the clinical features of this unique phenotype--unilateral VS with other NF2-related tumors.

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Periventricular lucency on computed tomography associated with hydrocephalus: what is the cause?. Frequently, physicians gave other diagnoses, ordered unnecessary diagnostic tests, and ordered treatments other than the discontinuation of ACE inhibitor therapy. Similarly, UV induced protein inactivation revealed that laminin present in the inner retina did not provide a guidance cue. We conclude that rhIL-4 has an antiproliferative effect on the growth of some NSCLC in vitro and in vivo. One might analyze the issue of courtship of the paying patient as a problem of distributive justice. Nonetheless, a growing number of studies are rapidly identifying transcriptional biomarkers in peripheral blood cells that may function as biomarkers of disease, evidence of pharmacodynamic effect, or even predictors of clinical outcomes and risk of toxicity. To facilitate the molecular diagnosis of uncharacterized patients with HPFH and delta beta thalassemia, oligonucleotide primers have been designed to enzymatically amplify deletion-specific products for nine known deletions, which include those responsible for HPFH-1, HPFH-2, HPFH-3, Spanish (delta beta)zero thalassemia, hemoglobin (Hb) Lepore, Sicilian (delta beta)zero thalassemia, Chinese G gamma(A gamma delta beta)zero thalassemia, Asian-Indian inversion-deletion G gamma(A gamma delta beta)zero thalassemia, and Turkish inversion-deletion (delta beta)zero thalassemia. Sophisticated animal experiments provided important data about enhanced arterial inflow and occlusion of venous outflow which cause and maintain erection of the penis.

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