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Bimode (Glipizide)

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Bimode is a medicine used to treat type 2 diabetes mellitus in adults. It belongs to a group of medicines called sulfonylureas and helps control blood sugar levels in people with diabetes. This helps to prevent serious complications of diabetes like kidney damage and blindness.

Other names for this medication:
Actizide, Bimode, Diabizide, Diacon, Diaglip, Dibimet, Dibizide, Glanase, Glibetic, Glimet, Glipicon, Glipicontin, Glipid, Glipimet, Glipiriv, Glipixor, Glipized, Glipizide, Glipmet, Glucolip, Gluconil, Glumin, Glunas, Glycind, Glydal, Glylin, Glynase, Glysar, Glysin, Glytop, Glyzip, Gpzide, Metaglez, Swiglip, Tranase, Zidmin

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Also known as: Glipizide


Bimode is a medication consists in a class of drugs called sulfonylureas. Bimode is used to treat type 2 diabetes. Bimode may be used along with diet, exercise and insulin therapy. Bimode is also known as Glipizide, Glytop SR. Bimode works by controlling blood sugar levels in your organism. Generic name of Bimode is Glipizide. Brand names of Bimode are Bimode, Bimode XL.


Usual Adult Dose of Glucotrol for Diabetes Type 2:

Immediate release: Initial dose: 5 mg orally once a day, 30 minutes before breakfast Maintenance dose: Up to 40 mg in divided doses 30 minutes before a meal of adequate caloric content. Doses may be increased in intervals of 2.5 to 5 mg a day according to blood glucose response. Maximum single dose: 15 mg Maximum daily dose: 40 mg Comments: -At least several days should elapse between titration steps. -If response to a single dose is not satisfactory, dividing that dose may prove effective. Extended Release: Initial dose: 5 mg orally once a day, 30 minutes before breakfast Maintenance dose: 5 to 10 mg orally once a day Maximum daily dose: 20 mg Patients receiving immediate release may be switched safely to extended release tablets once-a-day at the nearest equivalent total daily dose, or titrate to the appropriate extended release dose starting with 5 mg once daily. Combination use: -When adding other blood-glucose-lowering agents to the extended release tablets, the agent should be initiated at the lowest recommended dose. Observe for hypoglycemia. -When adding extended release tablets to other blood-glucose-lowering agents, Glucotrol XL extended release can be initiated at 5 mg. Start at a lower dose in patients that are more sensitive to hypoglycemia. When transferring patients from insulin to glipizide, the following general guidelines should be considered: -For patients with daily insulin doses of 20 units or less: Discontinue insulin and begin glipizide at usual dosages. -For patients with daily insulin doses greater than 20 units: Insulin dose should be reduced by 50% and glipizide therapy may begin at usual dosages. Comments: -Several days should elapse between titration steps. -Subsequent reductions in insulin dosage should depend on individual patient response. -During the insulin withdrawal period, the patient should test urine samples for sugar and ketone bodies at least three times daily. -Some patients receiving greater than 40 units of insulin daily may need to consider hospitalization during the transition period. Patients Receiving Other Oral Hypoglycemic Agents: -When transferring from longer half-life sulfonylureas: Observe for 1 to 2 weeks for hypoglycemia. -Glucotrol XL extended release coadministered with colesevelam: Glipizide should be administered at least 4 hours prior to colesevelam. Use: Adjunct to diet and exercise to improve glycemic control in adults with type 2 diabetes mellitus.

Usual Geriatric Dose of Glucotrol for Diabetes Type 2:

Initial dose: 2.5 mg orally once a day 30 minutes before breakfast Comments: -Maintenance dosing should be conservative to avoid hypoglycemic reactions. -Doses can be adjusted with caution taking into account the degree of hepatic, renal, or cardiac function, and the concomitant disease or other drug therapy. Use: Adjunct to diet and exercise to improve glycemic control in adults with type 2 diabetes mellitus.

Detailed Glucotrol dosage information


Seek emergency medical attention or call the Poison Help line at 1-800-222-1222. A glipizide overdose can cause life-threatening hypoglycemia.

Symptoms of severe hypoglycemia include extreme weakness, blurred vision, sweating, trouble speaking, tremors, stomach pain, confusion, and seizure (convulsions).


Store the medicine in a closed container at room temperature, away from heat, moisture, and direct light. Keep from freezing.

Keep out of the reach of children.

Do not keep outdated medicine or medicine no longer needed.

Ask your healthcare professional how you should dispose of any medicine you do not use.

Detailed Glucotrol XL dosage information

Side effects

The most common side effects associated with Bimode are:

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Appropriate studies performed to date have not demonstrated geriatric-specific problems that would limit the usefulness of glipizide in the elderly. However, elderly patients are more likely to have age-related heart, liver, or kidney problems which may require caution in patients receiving glipizide.

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Gastrointestinal function should be taken into consideration before using enteral nutrition, and enteral nutrition should not be the sole stress ulcer prophylactic therapy. It is a rare histiocytic disease of unknown etiology that is characterized pathologically by xanthogranulomatous infiltrates of multiple organs. Moreover, our theory allows us to explain previous findings, such as the deviation from the Lyapunov decay rate in cases where the classical finite-time instability is nonuniform in phase space. An 8.2 kb DNA segment from chromosome XIV carries the RPD3 and PAS8 genes as well as the Saccharomyces cerevisiae homologue of the thiamine-repressed nmt1 gene and a chromosome III-duplicated gene for a putative aryl-alcohol dehydrogenase. The CP approach offers a more extensive working area than the RS for lesions involving the anterolateral surface of the brainstem, but not for petroclival lesions. Electromyographic signs of shoulder muscular fatigue were studied in abduction and forward flexion at right angle in the shoulder joint for seven ticlid cost subjects. The regional variability of the capillary network from arterial to venous end also contributes to the data spread. Stunning of the left atrium after conversion of atrial fibrillation: predictor for maintenance of sinus rhythm?.

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A trichloroethylene risk assessment using a Monte Carlo analysis of parameter uncertainty in conjunction with physiologically-based pharmacokinetic modeling. This indicated the necessity to consider such babies as a group at risk of developing neonatal complications. Growth, bone mass, and vitamin D status of Chinese adolescent girls 3 y after withdrawal of milk supplementation. Spontaneous cerebrospinal fluid rhinorrhea through the cribriform plate fistula cured by endonasal surgery: transseptal submucoperiosteal obliteration of the olfactory cleft. In a further bilateral ovarian tumour a partial LOH affecting 17q22-q25 was present in one ovary only. We evaluated the reliability hospital discharge register data in the assessment of acute myocardial infarction trends. Thus, at the moment, the formation of capsular bend at the posterior optic edge appears to be the decisive criterion to evaluate the buy zovirax cream usa influence of IOL material and design on the development of PCO.

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Right atrial angiosarcoma causing a coronary artery fistula: diagnosis by transesophageal echocardiography. These observations indicate that recN is involved in the induced excision of Tn10. The participants were followed over a 10-year period by word- and non-word-reading tests and tests of cognitive ability. In contrast, no inhibition of involucrin or K1 expression was observed at the protein level at the same retinoic acid concentration. Effects of adrenaline and noradrenaline on the cardiovascular system. These results indicate that PLA has a protective effect likely involving elicitation of a cytokine response in vivo against fungal infections. Syphilitic coronary obstruction: correction under artificial heart-lung and profound hypothermia. Wnt2b inhibits differentiation of retinal progenitor cells in the absence of Notch activity by downregulating the expression of proneural genes. Radial double immunodiffusion experiments using antiserum to C1q, C1r, and C1s on HFf-treated serum demonstrated the dissociation of the C1 trimolecular complex, with concomitant reduction of C1r antigenicity that is indicative of C1 activation. The granules were similar to secretory vesicles in size, but were more irregular in shape.

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A course of deep x-ray therapy was then delivered to the mandibular lesion. From 1995 to 2003, 36 patients with penile and scrotal EMPD were treated and followed up. The pharmacokinetic profile of the best analog and the in vivo efficacy in an ovalbumin-induced bronchoconstriction assay in conscious guinea pigs are reported. In addition, recombinant IL-6 induced in vitro growth of TEC, demonstrating that IL-6 is a possible autocrine growth factor for these cells. However, in some of the datasets analyzed, the Lhcb4 and Lhcb6 genes as well as an Arabidopsis gene not present in poplar (Lhcb2.3) exhibited minor differences to the main cooperative Lhc gene expression pattern. When investigated under Ca intravenous drop infusion in 20 recipients and 14 healthy subjects, Ca tissue binding confirmed its physiochemical nature and revealed in one third of the recipients impaired buffer capacity of the tissues as well as Ca retention and release rates. Coronary vascular and myocardial effects of substance P in hypercholesterolemic rabbits.

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Brain met-enkephalin immunostaining after subacute and subchronic exposure to benzene. Immunohistochemical tumor diagnosis--possibilities and limitations. Influence of high temperature and humidity on the reproductive performance of swine. The cerebrospinal fluid primidone pill analysis showed discrete changes in the cell differential count. Pododermal microvascularization has been suggested to play a key role in the physiological function of the bovine claw and in the pathogenesis of claw diseases. Identification of a nonsense mutation in the granulocyte-colony-stimulating factor receptor in severe congenital neutropenia. H5N1/97-infected MDMs exhibited the strongest induction of apoptosis in Jurkat T cells, and it could be reduced by TRAIL-receptor 2 blocking antibody.

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