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Amvik (Atenolol)

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Amvik is a selective beta blocker. It works by blocking specific (natural) compounds that may bind to receptors on the heart. This slows down your heart rate so your heart doesn't have to work as hard.

Other names for this medication:
Adbeta, Altoma, Amcard, Amchek, Amdonex, Amfast, Amlocal, Amlodac, Amlong, Amlopres, Amlosafe, Amone, Amopine, Ampin, Amvik, Amzen, Andinx, Angicam, Angil, Angipres, Atemarc, Betatrop, Cardipin, Cardules, Defidin, Dilcare, Joglit, Nusar, Obeta, Presolar, Primodil, Telol, Winten, Ziblok

Similar Products:
Indocin, Nimotop, Cycrin, Omnicef, Arava, Dulcolax, Prevacid, Hytrin, Adalat

Also known as: Atenolol


Tenormin (atenolol) is a beta-blocker that affects the heart and circulation (blood flow through arteries and veins).

Tenormin is used to treat angina (chest pain) and hypertension (high blood pressure).

Tenormin is also used to lower the risk of death after a heart attack.


You should take it by mouth with meals or without it. Take Amvik every day at the same time and remember that its dosage depends on patient's health state. Elderly people: Starting normal dosage is 25-50 mg a day. Adults: For angina treatment: normal starting dosage is 50 mg a day. Max dosage is 100 mg a day. For heart attack treatment: normal starting dose is 25 mg a day. Max dosage is 50 mg a day. For hypertension treatment: normal starting dose is 50 mg a day. Max dose is 100 mg a day. If you want to achieve most effective results do not stop taking Amvik suddenly.


Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Overdose symptoms may include extreme weakness or lack of energy, very slow heart rate, shortness of breath, or fainting.


Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Amvik are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


In deciding to use a medicine, the risks of taking the medicine must be weighed against the good it will do. This is a decision you and your doctor will make. For this medicine, the following should be considered:

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Comparison of muscle phosphofructokinase from euthermic and hibernating Jaculus orientalis. Therefore, finding ways to improve VO2max and reduce blood pressure it is of vital importance to public health. Characterization of hematopoietic intracellular protein tyrosine phosphatases: description of a phosphatase containing an SH2 domain and another enriched in proline-, glutamic acid-, serine-, and threonine-rich sequences. Ultrasound studies were assessed for lesion shape, size, echogenicity, heterogeneity, and vascularity. SNPs (rs1165205, rs3733591, rs6855911, rs2231142, rs333049) were genotyped neurontin generic names by allele-specific polymerase chain reaction (PCR) and analyzed with SPSS 16.0. The release of prion protein from platelets during storage of apheresis platelets. This article highlights the importance of core body temperature and outcomes of AAA repair. Both populations became non-culturable within 30 min, with T90s of 15 min and 25 min, respectively. GP125/CD98 is a heterodimeric 125-kDa glycoprotein, which consists of an 85-kDa heavy chain (hc) and a 40-kDa light chain (lc), and is strongly expressed on the cell surface of various tumor cells, irrespective of their tissue of origin.

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Chlordiazepoxide (CDPX) enhanced the rate of chloride exchange mediated by the major GABAA receptor found on sealed native membrane vesicles from rat cerebral cortex. The antipeptide antibodies recognize VP2 and amino acid sequence Asp-His-Lys (193-195) in VP1 at different degrees of denaturation. Pyogenic Arthritis, Pyoderma Gangrenosum, Acne, Suppurative Hidradenitis (PA-PASH) Syndrome: An Atypical Presentation of a Rare Syndrome. Involvement of endogenously generated opioid substances in the control of breathing of patients with COPD is further suggested by this study. Lack of appropriate pharmacological tools for probing the 5HT-1a and 5HT-1b binding sites has made it difficult to definitively determine whether these binding sites are coupled to biochemical effector systems or mediate any of the physiological responses to 5HT agonists.

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