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Amozen (Atenolol)

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Amozen is a beta blocker that works specifically on the heart. It works by slowing down the heart rate and makes the heart more efficient at pumping blood around the body. Amozen is also used to relieve the symptoms caused by an overactive thyroid gland.

Other names for this medication:
Adbeta, Aloten, Altol, Amfast, Amlip, Amlobeat, Amlocom, Amloman, Amlong, Amlop, Amlorid, Amtas, Amvik, Amzen, Angipres, Angizaar, Atekind, Atenij, Atenodip, Atepose, Atoli, Betanif, Beten, Carden, Cardipin, Cardpin, Defidin, Dibeta, Espin, Hynorm, Latenol, Loten, Nifetex, Nifetolol, Nipidol, Noldepin, Novaten, Obeta, Odinol, Pertenol, Primodil, Quinoten, Systonorm, Teenol, Tenodipin, Tentoric, Vascodil, Windep

Similar Products:
Requip, Viagra, Ilosone, Desyrel, Mircette, Compazine

Also known as: Atenolol


Amozen is created by pharmacy specialists to combat angina, heart attack, high blood pressure, anxiety attacks and migraine. Target of Amozen is to influence circulation. Amozen is also known as Atenolol, Hipres, Atendol, Altol, Atelol, Antipressan, Atenix, Ateno, Betacard, Blokium, Catenol, Corotenol, Notenol, Oraday. Amozen acts as an anti-angina remedy. Amozen operates by controlling and decreasing the strength of heart contractions and, thus, influencing circulation. Amozen is a beta-blocker. You can use Amozen with other anti-high blood pressure medication. Generic name of Amozen is Atenolol. Brand name of Amozen is Amozen.


You should take it by mouth with meals or without it. Take Amozen every day at the same time and remember that its dosage depends on patient's health state. Elderly people: Starting normal dosage is 25-50 mg a day. Adults: For angina treatment: normal starting dosage is 50 mg a day. Max dosage is 100 mg a day. For heart attack treatment: normal starting dose is 25 mg a day. Max dosage is 50 mg a day. For hypertension treatment: normal starting dose is 50 mg a day. Max dose is 100 mg a day. If you want to achieve most effective results do not stop taking Amozen suddenly.


If you overdose Amozen and you don't feel good you should visit your doctor or health care provider immediately. Symptoms of Amozen are low blood sugar, slow heartbeat, heart failure, slowness, low blood pressure, wheezing.


Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Amozen are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


You should not use Tenormin if you are allergic to atenolol, or if you have:

a serious heart condition such as "AV block" (second or third degree); very slow heartbeats; or heart failure.

To make sure Tenormin is safe for you, tell your doctor if you have:

congestive heart failure; coronary artery disease (hardened arteries); asthma, bronchitis, emphysema; diabetes; overactive thyroid; liver or kidney disease; pheochromocytoma (tumor of the adrenal gland); peripheral vascular disease such as Raynaud's syndrome; or allergies (or if you are undergoing allergy treatments or skin-testing).

Using Tenormin during pregnancy could harm the unborn baby. Tell your doctor if you are pregnant or if you become pregnant while using this medicine.

Atenolol can pass into breast milk and may harm a nursing baby. Tell your doctor if you are breast-feeding a baby.

Tenormin is not approved for use by anyone younger than 18 years old.

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Wnt signaling by EGL-20/Wnt controls the migration of the descendants of the QL neuroblast by regulating the expression the Hox gene mab-5. Analysis of nitrite in adulterated urine samples by capillary electrophoresis. The 4A6-reactive antigen was found on the basement membrane and luminal surface of the epithelial cells in the caput epididymis of BALB/c male mice as well as in the proximal corpus epididymis, the cauda epididymis, and the vas deferens. vitamin D3 at commencement of dialysis we investigated whether 1 alpha (OH)D3 or 1,25(OH)2D3 acutely modifies the responsiveness of the parathyroid gland to the suppressive effect of increased serum calcium. In cognitive tasks, error commission is usually followed by a performance characterized by post-error slowing (PES) and post-error improvement of accuracy (PIA). It appears that high resolution spinal MR, using its various tissue parameters provides no new insights into possible recurrence of a disc prolapse. Inhibition of phosphoinositide metabolism in human polymorphonuclear leukocytes by S-adenosylhomocysteine. Analysis of these interactions revealed that a single ECM protein, thrombospondin, in conjunction with a single cytokine (e.g., c-kit ligand), constitutes a developmental signal that synergistically modulates hematopoietic stem cell function. Fluorescence studies of the molecular conformation of alpha-lactalbumin.

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Effect of portacaval shunt and arterialization of the liver on bile acid metabolism. The search identified eight guidelines that had partial relevance to the topic of the present guideline and thirty-eight primary studies. The advent of high-sensitivity flow cytometry techniques, developed to monitor disease in CLL patients undergoing treatment, has allowed accurate enumeration of sub-clinical levels of monoclonal B-cells with a CLL phenotype in healthy individuals from the general population and CLL families. Histopathologic examination during the period of viral shedding revealed a mild, nonsuppurative enteritis. The synthesis of total nuclear proteins and desoxyribonucleic acid during the preparation of mitosis. Some features of acute CRPS (vasodilatation, swelling, pain) indicate a localized inflammatory process. Descending inhibition in the rabbit proximal colon, evoked by distension, was studied in vivo by recording extracellularly electrical activity from pressure electrodes placed on the serosa. The putative forkhead transcription factor FOXL2 is mutated in blepharophimosis/ptosis/epicanthus inversus syndrome. Cbfa1, a transcription factor of the runt family, was recently shown to be a key regulator in skeletal development in mammals.

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